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dc.contributor.authorSoriano, Vicente
dc.contributor.authorGanado-Pinilla, Pilar
dc.contributor.authorSánchez-Santos, Miguel
dc.contributor.authorBarreiro, Pablo
dc.date2020
dc.date.accessioned2021-05-14T08:10:29Z
dc.date.available2021-05-14T08:10:29Z
dc.identifier.issn1139-6121
dc.identifier.urihttps://reunir.unir.net/handle/123456789/11339
dc.description.abstractThe clinical spectrum of “Severe Acute Respiratory Syndrome Coronavirus type 2” (SARS-CoV-2) infection is wider than initially thought. The coronavirus does not establish a chronic cellular infection, in contrast with HIV or the hepatitis B virus, that keeps their genomes, respectively, as proviruses integrated within the chromosomes or as episomes (Soriano et al. J Antimicrob Chemother 2014).es_ES
dc.language.isoenges_ES
dc.publisherAIDS Reviewses_ES
dc.relation.ispartofseries;vol. 22, nº 4
dc.relation.urihttps://pubmed.ncbi.nlm.nih.gov/33401287/es_ES
dc.rightsopenAccesses_ES
dc.subjectamphotericin Bes_ES
dc.subjectC reactive proteines_ES
dc.subjecthaptoglobines_ES
dc.subjectimmunoglobulin Ges_ES
dc.subjectScopus(2)es_ES
dc.titleHot news: Unveiling long COVID-19 diseasees_ES
dc.typeotheres_ES
reunir.tag~ARIes_ES
dc.identifier.doihttps://doi.org/10.24875/AIDSRev.M20000039


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